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10.1038/s41593-022-01221-3 [DOI] [PMC free article] [PubMed] [Google Scholar] Sanaeifar F., Pourranjbar S., Pourranjbar M., Ramezani S., Mehr S

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The pivotal role of FDX1 in this pathway is underscored by experimental evidence that genetic ablation of FDX1 confers significant protection against DOX-induced cardiotoxicity in mice, preserving the left ventricular ejection fraction [76]