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Mutations of the EPHA2 receptor tyrosine kinase gene cause autosomal dominant congenital cataract

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The pathological observation of airway remodeling within the epithelial airway is a significant indication of structural changes including loss of epithelial integrity leading to epithelial-mesenchymal transition (EMT), destruction of ciliated cells, a decrease in EMT-related epithelial markers, and an increase in EMT-related mesenchymal markers, goblet cell and mucous gland hyperplasia and metaplasia, an increase in the production of extracellular matrix (ECM) components like fibronectin, collagen types III and V, and MMP-9 within the reticular basement membrane, and upregulation of growth factor release and the overexpression of receptors, such as epidermal growth factor receptors (EGFR) (80–83)